Please note: In 2003, the CTF updated its Grades of Recommendations to include an "I Recommendation" for situations where insufficient evidence exists to allow a recommendation to be made. (Formerly, these situations were captured under a "C Recommendation".) This change is not retroactive, and all "C Recommendations" made prior to 2003 have not been reevaluated in light of the new "I" recommendation grade. For a discussion of these recommendation grades, please link to the 2003 article in the Canadian Medical Association Journal here.
There is currently little evidence from published clinical research to suggest that routine iron supplementation during pregnancy improves clinical outcomes for the mother, fetus or newborn. The evidence is insufficient to recommend for or against routine iron supplementation during pregnancy (C Recommendation).
These conclusions apply only to routine iron supplementation and do not pertain to the selection of iron containing foods as part of a healthful pregnancy diet, the use of screening tests to detect anemia during pregnancy, the proper clinical evaluation of the causes of anemia when it is discovered, or the selective use of iron supplements in pregnant women with documented iron deficiency anemia. Prevention of iron deficiency anemia in infants is addressed in Chapter 23.
Because relevant effectiveness data are inadequate, clinicians must use individual judgement in determining how to counsel pregnant women about dietary intake of iron containing foods and iron supplements and in deciding whether and how to screen women for anemia and iron deficiency.
For years mineral and vitamin supplements have been prescribed routinely to pregnant women as a normal part of prenatal care. These supplements are often prescribed as preparations that include 25-65 mg of elemental iron, along with other minerals (e.g., calcium, zinc, magnesium, copper) and vitamins. However, few studies have examined the clinical effectiveness of prenatal vitamin preparations as a group.
Although studies of male workers have demonstrated low productivity among iron deficient men, few studies of the health effects of iron deficiency have included women, let alone pregnant women. A Swedish survey of 1,462 women compared the complaints of 82 anemic women (Hgb <120 g/L) with those of non-anemic women and found no difference in the incidence of reported infections, fatigue, sleeping difficulties, headache, or work absenteeism.<2> Anemic women were significantly more likely to report low work productivity than non-anemic women (10% vs. 5%). Physical symptoms of anemia are generally unapparent unless hemoglobin values fall below 70-80 g/L.
Cross-sectional and longitudinal observational studies (grade II-2 evidence) in the U.S. and Europe have demonstrated that even mild to moderate anemia can be associated with adverse obstetrical outcomes, including preterm delivery, low birth weight and fetal death.<3,4> However, most of the studies do not control for other factors that can cause low birth weight and prematurity (e.g., poor nutrition, smoking), making it unclear whether anemia and iron deficiency are merely associated with these variables rather than having a direct influence on pregnancy outcomes.
However, most studies suggest that pregnant women who are iron deficient are no more likely to give birth to iron deficient newborns than women who have adequate iron stores.<5> Nor is there direct evidence that pregnant women who take iron supplements give birth to infants or children with improved mental or psychomotor performance.
The review by the U.S. Preventive Services Task Force of the evidence for effectiveness focused solely on the ability of iron supplements to improve clinical outcomes in either the mother or newborn (e.g. low birth weight, preterm birth). The biological effectiveness of iron supplements in changing non-clinical outcomes (e.g. hematocrit, hemoglobin, ferritin levels) was not reviewed. This chapter will focus only on studies conducted in industrialized countries.
In a prospective, controlled cohort study in Sweden, Kullander and Kallen collected data on 6,376 women in Malm ¨ o in 1963-1965.<6> They found that women who took iron and vitamin supplements were significantly less likely to give birth before 38 weeks (6-9% of births) than women who did not use such supplements (11-13% of births). The birth weight of boys (but not girls) was significantly higher in women who took iron and vitamins than in those who took no supplements. However, without proper control for confounding variables, it is difficult to know whether women who took iron supplements had other characteristics (e.g. healthier lifestyle) that reduced their risk of adverse outcomes. Conversely, a Dutch prospective study indicated no association between low maternal hemoglobin and adverse perinatal outcomes.<7>
The strongest evidence on which to evaluate the effectiveness of routine iron supplementation comes from randomized controlled trials. Most clinical trials of iron supplementation have not demonstrated significant improvements in maternal or neonatal outcomes. Sample sizes in these trials are small, and thus statistical power is generally inadequate to prove that iron supplementation is ineffective. A quasi-experimental study in India reported improved birth weights with supplementation but that may have been confounded by improper randomization.<8> A Scottish randomized controlled trial of 3,600 patients found no difference in the incidence of a wide range of adverse obstetrical outcomes between those receiving iron and those receiving placebo.<9> In a randomized controlled trial with 3,000 women, Hemminki and Rimpela compared routine versus selective iron supplementation.<10> The "routine" group was advised to take 100 mg of elemental iron daily beginning by the 17th week of gestation, while the "selective" group was advised to take iron only if certain hematologic parameters were present. Women in the "selective" group were more likely to report poor overall health, to require transfusion and operative delivery, and to have newborns with reduced gestational age at birth. The difference in gestational age was not clinically significant, however, and the authors conjectured that lack of blinding may have contributed to the higher complication rate in the selective group.
In 1988, the U.S. Surgeon Generals Report on Nutrition and Health concluded that iron supplementation is a "reasonable approach" to the prevention of iron deficiency and included pregnant women among the groups that "may need iron supplements." The report also recommended that pregnant women receive laboratory evaluation for anemia and nutritional advice on methods to ensure adequate iron intake and to enhance iron bioavailability from the diet.
In 1989, the U.S. Public Health Service Expert Panel on the Content of Prenatal Care recommended that health promotion activities during routine preconception and prenatal visits should include counselling on vitamin and iron supplementation "on indication", for women at risk. The evidence on which this recommendation was based was classified as "fair." The panel also recommended routine hemoglobin and hematocrit measurements.
In a major report in 1990 on nutrition during pregnancy, the Food and Nutrition Board of the Institute of Medicine recommended routine use of daily iron supplements (30 mg/day) after about the twelfth week of gestation, in conjunction with a well-balanced diet that contains enhancers of iron absorption (ascorbic acid, meat). It also recommended that either hemoglobin or hematocrit should routinely be determined at the first prenatal visit. The report recommended that anemia accompanied by a low serum ferritin concentration should be treated with 60-120 mg of ferrous iron daily until a normal hemoglobin concentration is reached, after which the dose should be lowered to 30 mg daily.
Although observational data (grade II-2 evidence) suggest that pregnant women with anemia (hemoglobin less than 100 g/L) are at increased risk of preterm birth, low birth weight, or other adverse outcomes, it is unclear from such evidence whether anemia is responsible for these outcomes and whether they can be prevented through iron supplementation. Similarly, it is unclear whether iron supplementation during pregnancy can reduce the incidence of iron deficiency in infants, a condition that has been associated with delayed psychomotor development. Although iron supplementation can improve maternal hematologic indices, controlled clinical trials (grade I and II-1 evidence) have failed to demonstrate that iron supplementation or changes in hematologic indices actually improve clinical outcomes for the mother or newborn.
These conclusions apply only to routine iron supplementation and do not pertain to the selection of iron containing foods as part of a healthful pregnancy diet, the use of screening tests to detect anemia during pregnancy, the proper clinical evaluation of the causes of anemia when it is discovered, or the selective use of iron supplements in pregnant women with documented iron deficiency anemia.
Because relevant effectiveness data are inadequate, clinicians must use individual judgement in determining how to counsel pregnant women about dietary intake of iron-containing foods and iron supplements and in deciding whether and how to screen women for anemia and iron deficiency.
Link to Structured Abstract of this review
Link to Summary Table of this review
Link to Selected References list of this review
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© 1994 Minister of Supply and Services Canada.
Last modified March 27, 1998.